Gary M. Wilson
Credentials: Chemistry Ph.D., Spring 2020 (Coon)
Hometown: Boxford, MA
- Professor Joshua J. Coon
- Application and development of mass spectrometry technologies to enable biological investigations
- Future Plans
- After graduation, I will be starting a chemoproteomics position at a biotech company in San Diego. I thank the Biotechnology Training Program for fellowship during my graduate career.
View ORCID associated publications.
- Wilson, G. M., Blanco, R., Coon, J. J., & Hornberger, T. A. (2018). Identifying Novel Signaling Pathways: An Exercise Scientists Guide to Phosphoproteomics. Exercise and Sport Sciences Reviews, 46(2), 76–85. https://doi.org/110.1016/j.bbi.2017.04.008
- Tanimura, N., Liao, R., Wilson, G. M., Dent, M. R., Cao, M., Burstyn, J. N., Hematti, P., Liu, X., Zhang, Y., Zheng, Y., Keles, S., Xu, J., Coon, J. J., & Bresnick, E. H. (2018). GATA/Heme Multi-omics Reveals a Trace Metal-Dependent Cellular Differentiation Mechanism. Developmental Cell, 46(5), 581-594.e4. https://doi.org/10.1016/j.devcel.2018.07.022
- Niemi, N. M., Wilson, G. M., Overmyer, K. A., Vögtle, F.-N., Lohman, D. C., Schueler, K. L., Attie, A. D., Meisinger, C., Coon, J. J., & Pagliarini, D. J. (2019). Pptc7 is an essential phosphatase for promoting mammalian mitochondrial metabolism and biogenesis. Nature Communications, 10, 3197. https://doi.org/10.1101/426247
- Leung, K. K., WIlson, G. M., Kirkemo, L. L., Riley, N. M., Coon, J. J., & Wells, J. A. (2020). Broad and thematic remodeling of the surface glycoproteome on isogenic cells transformed with driving proliferative oncogenes. Proceedings of the National Academy of Sciences, 117(14). https://doi.org/10.1017/CBO9781107415324.004
I graduated from Wake Forest University in Winston-Salem, NC in 2010 and spent some time working in the Boston biotech sector before coming to graduate school. In 2015, I joined Pf. Joshua Coon’s lab as part of the chemical biology path. My research has centered on improving and applying glycoproteomic methods to study the cell surface changes that occur in consequence to oncogenic transformation.
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